CCDC88B is required for pathogenesis of inflammatory bowel disease-Reference-Cited by-同舟云学术

CCDC88B is required for pathogenesis of inflammatory bowel disease

Published:2017-10-13 Issue:1 Volume:8 Page:
ISSN:2041-1723
Container-title:Nature Communications
language:en
Short-container-title:Nat Commun

Author:

Fodil Nassima,Moradin Neda,Leung Vicki,Olivier Jean-Frederic,Radovanovic Irena^ORCID,Jeyakumar Thiviya,Flores Molina Manuel,McFarquhar Ashley,Cayrol Romain,Bozec Dominique,Shoukry Naglaa H.,Kubo Michiaki,Dimitrieva Julia,Louis Edouard,Theatre Emilie,Dahan Stephanie,Momozawa Yukihide,Georges Michel,Yeretssian Garabet,Gros Philippe

Publisher

Springer Science and Business Media LLC

Subject

General Physics and Astronomy,General Biochemistry, Genetics and Molecular Biology,General Chemistry

Link

http://www.nature.com/articles/s41467-017-01381-y.pdf

Reference41 articles.

1. Jostins, L. et al. Host-microbe interactions have shaped the genetic architecture of inflammatory bowel disease. Nature 491, 119-124 (2012).

2. Liu, J. Z. et al. Association analyses identify 38 susceptibility loci for inflammatory bowel disease and highlight shared genetic risk across populations. Nat. Genet. 47, 979–986 (2015).

3. Kaplan, G. G. The global burden of IBD: from 2015 to 2025. Nat Rev Gastroenterol. Hepatol. 12, 720–727 (2015).

4. Fodil, N., Langlais, D. & Gros, P. Primary Immunodeficiencies and Inflammatory Disease: A Growing Genetic Intersection. Trends Immunol. 37, 126–140 (2016).

5. Kennedy, J. M. et al. CCDC88B is a novel regulator of maturation and effector functions of T cells during pathological inflammation. J. Exp. Med. 211, 2519–2535 (2014).

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