Intron retention and nuclear loss of SFPQ are molecular hallmarks of ALS-Reference-Cited by-同舟云学术

Intron retention and nuclear loss of SFPQ are molecular hallmarks of ALS

Published:2018-05-22 Issue:1 Volume:9 Page:
ISSN:2041-1723
Container-title:Nature Communications
language:en
Short-container-title:Nat Commun

Author:

Luisier Raphaelle,Tyzack Giulia E.^ORCID,Hall Claire E.,Mitchell Jamie S.,Devine Helen,Taha Doaa M.,Malik Bilal,Meyer Ione,Greensmith Linda,Newcombe Jia,Ule Jernej,Luscombe Nicholas M.^ORCID,Patani Rickie

Publisher

Springer Science and Business Media LLC

Subject

General Physics and Astronomy,General Biochemistry, Genetics and Molecular Biology,General Chemistry

Link

http://www.nature.com/articles/s41467-018-04373-8.pdf

Reference69 articles.

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3. Tian, B. & Manley, J. L. Alternative polyadenylation of mRNA precursors. Nat. Rev. Mol. Cell. Biol. 18, 18–30 (2017).

4. Nilsen, T. W. & Graveley, B. R. Expansion of the eukaryotic proteome by alternative splicing. Nature 463, 457–463 (2010).

5. Buckley, P. T. et al. Cytoplasmic intron sequence-retaining transcripts can be dendritically targeted via ID element retrotransposons. Neuron 69, 877–884 (2011).

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