PPM1D gene amplification and overexpression in breast cancer: a qRT-PCR and chromogenic in situ hybridization study-Reference-Cited by-同舟云学术

PPM1D gene amplification and overexpression in breast cancer: a qRT-PCR and chromogenic in situ hybridization study

Published:2010-06-11 Issue:10 Volume:23 Page:1334-1345
ISSN:0893-3952
Container-title:Modern Pathology
language:en
Short-container-title:Mod Pathol

Author:

Lambros Maryou B,Natrajan Rachael,Geyer Felipe C,Lopez-Garcia Maria A,Dedes Konstantin J,Savage Kay,Lacroix-Triki Magali,Jones Robin L,Lord Christopher J,Linardopoulos Spiros,Ashworth Alan,Reis-Filho Jorge S

Publisher

Springer Science and Business Media LLC

Subject

Pathology and Forensic Medicine

Link

http://www.nature.com/articles/modpathol2010121.pdf

Reference43 articles.

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2. Bulavin DV, Demidov ON, Saito S, et al. Amplification of PPM1D in human tumors abrogates p53 tumor-suppressor activity. Nat Genet 2002;31:210–215.

3. Bulavin DV, Phillips C, Nannenga B, et al. Inactivation of the Wip1 phosphatase inhibits mammary tumorigenesis through p38 MAPK-mediated activation of the p16(Ink4a)-p19(Arf) pathway. Nat Genet 2004;36:343–350.

4. Hu X, Stern HM, Ge L, et al. Genetic alterations and oncogenic pathways associated with breast cancer subtypes. Mol Cancer Res 2009;7:511–522.

5. Li J, Yang Y, Peng Y, et al. Oncogenic properties of PPM1D located within a breast cancer amplification epicenter at 17q23. Nat Genet 2002;31:133–134.

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