JAK2 Mutations are present in all cases of polycythemia vera
Author:
Publisher
Springer Science and Business Media LLC
Subject
Oncology,Cancer Research,Hematology
Link
http://www.nature.com/articles/2405047.pdf
Reference5 articles.
1. James C, Ugo V, Le Couédic J-P, Staerk J, Delhommeau F, Lacout C et al. A unique clonal JAK2 mutation leading to constitutive signalling causes polycythaemia vera. Nature 2005; 434: 1144–1148.
2. Verstovsek S, Silver RT, Cross NCP, Tefferi A . JAK2V617F mutational frequency in polycythemia vera: 100%, >90%, less? Leukemia 2006; 20: 2067.
3. Jones AV, Kreil S, Zoi K, Waghorn K, Curtis C, Zhang L et al. Widespread occurrence of the JAK2 V617F mutation in chronic myeloproliferative disorders. Blood 2005; 106: 2162–2168.
4. Chen Q, Lu P, Jones AV, Cross NCP, Silver RT, Wang YL . Amplification refractory mutation system (ARMS), a highly sensitive and simple PCR assay for the detection of JAK2V617F mutation in chronic myeloproliferative disorders. J Mol Diagn 2007; 9: 272–276.
5. Wang YL, Lee JW, Kui JS, Chadburn A, Cross NCP, Knowles DM et al. Evaluation of JAK2V617F in B- and T-cell neoplasms: identification of JAK2V617F mutation of undetermined significance (JMUS) in the bone marrow of three individuals. Acta Haematologica 2007; 118: 209–214.
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