Hyporesponsiveness to the anti-inflammatory action of interleukin-10 in type 2 diabetes-Reference-Cited by-同舟云学术

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Hyporesponsiveness to the anti-inflammatory action of interleukin-10 in type 2 diabetes

Published:2016-02-17 Issue:1 Volume:6 Page:
ISSN:2045-2322
Container-title:Scientific Reports
language:en
Short-container-title:Sci Rep

Author:

Barry Julianne C.,Shakibakho Soroush,Durrer Cody,Simtchouk Svetlana,Jawanda Kamaldeep K.,Cheung Sylvia T.,Mui Alice L.,Little Jonathan P.

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/srep21244.pdf

Reference41 articles.

1. Gregor, M. F. & Hotamisligil, G. S. Inflammatory mechanisms in obesity. Annu. Rev. Immunol. 29, 415–445 (2011).

2. Pickup, J. C. Inflammation and activated innate immunity in the pathogenesis of type 2 diabetes. Diabetes Care 27, 813–823 (2004).

3. Dasu, M. R., Devaraj, S., Zhao, L., Hwang, D. H. & Jialal, I. High glucose induces toll-like receptor expression in human monocytes: Mechanism of activation. Diabetes 57, 3090–3098 (2008).

4. Dasu, M. R. & Jialal, I. Free fatty acids in the presence of high glucose amplify monocyte inflammation via toll-like receptors. Am. J. Physiol. Endocrinol. Metabol. 300, E145–154 (2011).

5. Schilling, J. D. et al. Palmitate and lipopolysaccharide trigger synergistic ceramide production in primary macrophages. J. Biol. Chem. 288, 2923–2932 (2013).

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