DR5 and caspase-8 are dispensable in ER stress-induced apoptosis-Reference-Cited by-同舟云学术

DR5 and caspase-8 are dispensable in ER stress-induced apoptosis

Published:2017-04-14 Issue:5 Volume:24 Page:944-950
ISSN:1350-9047
Container-title:Cell Death & Differentiation
language:en
Short-container-title:Cell Death Differ

Author:

Glab Jason A,Doerflinger Marcel,Nedeva Christina,Jose Irvin,Mbogo George W,Paton James C,Paton Adrienne W,Kueh Andrew J,Herold Marco J,Huang David CS,Segal David,Brumatti Gabriella,Puthalakath Hamsa

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology,Molecular Biology

Link

http://www.nature.com/articles/cdd201753.pdf

Reference16 articles.

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2. Bravo R, Vicencio JM, Parra V, Troncoso R, Munoz JP, Bui M et al. Increased ER-mitochondrial coupling promotes mitochondrial respiration and bioenergetics during early phases of ER stress. J Cell Sci 2011; 124 (Pt 13): 2143–2152.

3. Kaufman RJ . Orchestrating the unfolded protein response in health and disease. J Clin Invest 2002; 110: 1389–1398.

4. Puthalakath H, O'Reilly LA, Gunn P, Lee L, Kelly PN, Huntington ND et al. ER stress triggers apoptosis by activating BH3-only protein Bim. Cell 2007; 129: 1337–1349.

5. Reimertz C, Kogel D, Rami A, Chittenden T, Prehn JH . Gene expression during ER stress-induced apoptosis in neurons: induction of the BH3-only protein Bbc3/PUMA and activation of the mitochondrial apoptosis pathway. J Cell Biol 2003; 162: 587–597.

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