Transcriptional dysregulation during myeloid transformation in AML
Author:
Publisher
Springer Science and Business Media LLC
Subject
Cancer Research,Genetics,Molecular Biology
Link
http://www.nature.com/articles/1210765.pdf
Reference78 articles.
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2. Barjesteh van Waalwijk van Doorn-Khosrovani SB, Erpelnick C, Meijer J, Oosterhoud S, van Putten WL, Valk PJ et al. (2003). Biallelic mutations in the CEBPA gene and low CEBPA expression levels as prognostic markers in intermediate-risk AML. Hematol J 4: 31–40.
3. Behre G, Singh SM, Liu H, Bortolin LT, Christopeit M, Radomska HS et al. (2002). Ras signaling enhances the ability of CEBPA to induce granulocytic differentiation by phosphorylation of serine 248. J Biol Chem 277: 26293–26299.
4. Bienz M, Ludwig M, Oppliger-Leibundgut E, Mueller BU, Solenthaler M, Fey MF et al. (2005). Risk assessment in patients with acute myeloid leukemia and a normal karyotype. Clin Cancer Res 11: 1416–1425.
5. Bullinger L, Dohner K, Bair E, Frohling S, Schlenk RF, Tibshirani R et al. (2004). Use of gene-expression profiling to identify prognostic subclasses in adult acute myeloid leukemia. N Engl J Med 350: 1605–1616.
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