Transcription factor TAFII250 promotes Mdm2-dependent turnover of p53
Author:
Publisher
Springer Science and Business Media LLC
Subject
Cancer Research,Genetics,Molecular Biology
Link
http://www.nature.com/articles/1210209.pdf
Reference34 articles.
1. Argentini M, Barboule N, Wasylyk B . (2001). The contribution of the acidic domain of MDM2 to p53 and MDM2 stability. Oncogene 20: 1267–1275.
2. Ashcroft M, Taya Y, Vousden KH . (2000). Stress signals utilize multiple pathways to stabilize p53. Mol Cell Biol 20: 3224–3233.
3. Blattner C, Hay TJ, Meek DW, Lane DP . (2002). Hypophosphorylation of Mdm2 augments p53 stability. Mol Cell Biol 22: 6170–6182.
4. Bottger A, Bottger V, Sparks A, Liu WL, Howard SF, Lane DP . (1997). Design of a synthetic Mdm2-binding mini protein that activates the p53 response in vivo. Curr Biol 7: 860–869.
5. Buchmann AM, Skaar JR, DeCaprio JA . (2004). Activation of a DNA damage checkpoint response in a TAF1-defective cell line. Mol Cell Biol 24: 5332–5339.
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