Neuronal expression of the transcription factor Gli1 using the Tα1 α-tubulin promoter is neuroprotective in an experimental model of Parkinson's disease-Reference-Cited by-同舟云学术

Neuronal expression of the transcription factor Gli1 using the Tα1 α-tubulin promoter is neuroprotective in an experimental model of Parkinson's disease

Published:2004-12 Issue:24 Volume:11 Page:1742-1752
ISSN:0969-7128
Container-title:Gene Therapy
language:en
Short-container-title:Gene Ther

Author:

Suwelack D,Hurtado-Lorenzo A,Millan E,Gonzalez-Nicolini V,Wawrowsky K,Lowenstein PR,Castro MG

Publisher

Springer Science and Business Media LLC

Subject

Genetics,Molecular Biology,Molecular Medicine

Link

http://www.nature.com/articles/3302377.pdf

Reference54 articles.

1. Mandel S et al. Neuroprotective strategies in Parkinson's disease: an update on progress. CNS Drugs 2003; 17: 729–762.

2. Kordower JH et al. Neurodegeneration prevented by lentiviral vector delivery of GDNF in primate models of Parkinson's disease. Science 2000; 290: 767–773.

3. Kirik D, Rosenblad C, Bjorklund A, Mandel RJ . Long-term rAAV-mediated gene transfer of GDNF in the rat Parkinson's model: intrastriatal but not intranigral transduction promotes functional regeneration in the lesioned nigrostriatal system. J Neurosci 2000; 20: 4686–4700.

4. Choi-Lundberg DL et al. Dopaminergic neurons protected from degeneration by GDNF gene therapy. Science 1997; 275: 838–841.

5. Castro MG et al. Gene therapy for Parkinson's disease: recent achievements and remaining challenges. Histol Histopathol 2001; 16: 1225–1238.

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