Bace1-dependent amyloid processing regulates hypothalamic leptin sensitivity in obese mice-Reference-Cited by-同舟云学术

Bace1-dependent amyloid processing regulates hypothalamic leptin sensitivity in obese mice

Published:2018-01-08 Issue:1 Volume:8 Page:
ISSN:2045-2322
Container-title:Scientific Reports
language:en
Short-container-title:Sci Rep

Author:

Meakin Paul J.^ORCID,Jalicy Susan M.,Montagut Gemma^ORCID,Allsop David J. P.,Cavellini Daniella L.,Irvine Stuart W.,McGinley Christopher,Liddell Mary K.,McNeilly Alison D.,Parmionova Karolina,Liu Yu-Ru,Bailey Charlotte L. S.,Dale J. Kim,Heisler Lora K.,McCrimmon Rory J.,Ashford Michael L. J.^ORCID

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/s41598-017-18388-6.pdf

Reference39 articles.

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3. Zhao, J. et al. Beta-site amyloid precursor protein cleaving enzyme 1 levels become elevated in neurons around amyloid plaques: implications for Alzheimer’s disease pathogenesis. J. Neurosci. 27, 3639–3649 (2007).

4. Janson, J. et al. Increased risk of type 2 diabetes in Alzheimer disease. Diabetes 53, 474–481 (2004).

5. Zhao, W. Q. & Townsend, M. Insulin resistance and amyloidogenesis as common molecular foundation for type 2 diabetes and Alzheimer’s disease. Biochim. Biophys. Acta. 1792, 482–496 (2009).

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