The effect of succinate on brain NADH/NAD+ redox state and high energy phosphate metabolism in acute traumatic brain injury
Author:
Publisher
Springer Science and Business Media LLC
Subject
Multidisciplinary
Link
http://www.nature.com/articles/s41598-018-29255-3.pdf
Reference37 articles.
1. Carpenter, K. L. H. et al. Systemic, Local, and Imaging Biomarkers of Brain Injury: More Needed, and Better Use of Those Already Established? Front. Neurol. 6, 1–20 (2015).
2. Nordström, C. H., Nielsen, T. H., Schalén, W., Reinstrup, P. & Ungerstedt, U. Biochemical indications of cerebral ischaemia and mitochondrial dysfunction in severe brain trauma analysed with regard to type of lesion. Acta Neurochir. (Wien). 158, 1231–1240 (2016).
3. Jalloh, I. et al. Focally perfused succinate potentiates brain metabolism in head injury patients. J. Cereb. Blood Flow Metab. 37, 2626–2638 (2017).
4. Williamson, D. H., Lund, P. & Krebs, H. A. The redox state of free nicotinamide-adenine dinucleotide in the cytoplasm and mitochondria of rat liver. Biochem. J. 103, 514–27 (1967).
5. Garnett, M. R. et al. Altered cellular metabolism following traumatic brain injury: a magnetic resonance spectroscopy study. J. Neurotrauma 18, 231–240 (2001).
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