Angiotensin II-induced hypertension in rats is only transiently accompanied by lower renal oxygenation
Author:
Funder
British Heart Foundation
EC | Seventh Framework Programme
Nierstichting
ZonMw
Publisher
Springer Science and Business Media LLC
Subject
Multidisciplinary
Link
http://www.nature.com/articles/s41598-018-34211-2.pdf
Reference56 articles.
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2. Nangaku, M. Chronic hypoxia and tubulointerstitial injury: a final common pathway to end-stage renal failure. J Am Soc Nephrol 17, 17–25, https://doi.org/10.1681/ASN.2005070757 (2006).
3. Fine, L. G., Bandyopadhay, D. & Norman, J. T. Is there a common mechanism for the progression of different types of renal diseases other than proteinuria? Towards the unifying theme of chronic hypoxia. Kidney Int Suppl 75, S22–26 (2000).
4. Welch, W. J., Baumgartl, H., Lubbers, D. & Wilcox, C. S. Nephron pO2 and renal oxygen usage in the hypertensive rat kidney. Kidney Int 59, 230–237, https://doi.org/10.1046/j.1523-1755.2001.00483.x (2001).
5. Manotham, K. et al. Evidence of tubular hypoxia in the early phase in the remnant kidney model. J Am Soc Nephrol 15, 1277–1288 (2004).
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