Critical roles of serotonin-oxytocin interaction during the neonatal period in social behavior in 15q dup mice with autistic traits
Author:
Publisher
Springer Science and Business Media LLC
Subject
Multidisciplinary
Link
http://www.nature.com/articles/s41598-018-32042-9.pdf
Reference39 articles.
1. Nakatani, J. et al. Abnormal behavior in a chromosome- engineered mouse model for human 15q11-13 duplication seen in autism. Cell 137, 1235–46 (2009).
2. State, M. & Levitt, P. The conundrums of understanding genetic risks for autism spectrum disorders. Nat Neurosci 14, 1499–06 (2011).
3. Takumi, T. & Tamada, K. CNV biology in neurodevelopmental disorders. Curr Opin Neurobiol 48, 183–192 (2018).
4. Tamada, K. et al. Decreased exploratory activity in a mouse model of 15q duplication syndrome; implications for disturbance of serotonin signaling. PLoS ONE 5, e15126, https://doi.org/10.1371/journal.pone.0015126 (2010).
5. Nakai, N. et al. Serotonin rebalances cortical tuning and behavior linked to autism symptoms in 15q11-13 CNV mice. Sci Adv 3, e1603001, https://doi.org/10.1126/sciadv.1603001 (2017).
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