RNAi screening identifies Trypanosoma brucei stress response protein kinases required for survival in the mouse
Author:
Publisher
Springer Science and Business Media LLC
Subject
Multidisciplinary
Link
http://www.nature.com/articles/s41598-017-06501-8.pdf
Reference51 articles.
1. Trindade, S. et al. Trypanosoma brucei parasites occupy and functionally adapt to the adipose tissue in mice. Cell Host Microbe 19, 837–848 (2016).
2. McCulloch, R. & Field, M. C. Quantitative sequencing confirms VSG diversity as central to immune evasion by Trypanosoma brucei. Trends Parasitol. 31, 346–349 (2015).
3. Matthews, K. R., McCulloch, R. & Morrison, L. J. The within-host dynamics of African trypanosome infections. Philos. Trans. R. Soc. B Biol. Sci. 370, 20140288 (2015).
4. Parsons, M., Worthey, E. A., Ward, P. N. & Mottram, J. C. Comparative analysis of the kinomes of three pathogenic trypanosomatids: Leishmania major, Trypanosoma brucei and Trypanosoma cruzi. BMC Genomics 6, 127 (2005).
5. Nett, I. R. E. et al. The phosphoproteome of bloodstream form Trypanosoma brucei, causative agent of African sleeping sickness. Mol. Cell. Proteomics 8, 1527–38 (2009).
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