Amyloid precursor protein drives down-regulation of mitochondrial oxidative phosphorylation independent of amyloid beta
Author:
Publisher
Springer Science and Business Media LLC
Subject
Multidisciplinary
Link
http://www.nature.com/articles/s41598-017-10233-0.pdf
Reference63 articles.
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2. Kogel, D., Deller, T. & Behl, C. Roles of amyloid precursor protein family members in neuroprotection, stress signaling and aging. Exp Brain Res 217, 471–479, doi: 10.1007/s00221-011-2932-4 (2012).
3. Corrigan, F. et al. The neuroprotective activity of the amyloid precursor protein against traumatic brain injury is mediated via the heparin binding site in residues 96-110. Journal of neurochemistry 128, 196–204, doi: 10.1111/jnc.12391 (2014).
4. Clarke, J. et al. Overexpression of APP provides neuroprotection in the absence of functional benefit following middle cerebral artery occlusion in rats. Eur J Neurosci 26, 1845–1852, doi: 10.1111/j.1460-9568.2007.05807.x (2007).
5. Ayton, S. et al. Ceruloplasmin and beta-amyloid precursor protein confer neuroprotection in traumatic brain injury and lower neuronal iron. Free Radic Biol Med 69, 331–337, doi: 10.1016/j.freeradbiomed.2014.01.041 (2014).
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