Ceruloplasmin and β-amyloid precursor protein confer neuroprotection in traumatic brain injury and lower neuronal iron-Reference-Cited by-同舟云学术

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Ceruloplasmin and β-amyloid precursor protein confer neuroprotection in traumatic brain injury and lower neuronal iron

Published:2014-04 Issue: Volume:69 Page:331-337
ISSN:0891-5849
Container-title:Free Radical Biology and Medicine
language:en
Short-container-title:Free Radical Biology and Medicine

Author:

Ayton Scott,Zhang Moses,Roberts Blaine R.,Lam Linh Q.,Lind Monica,McLean Catriona,Bush Ashley I.,Frugier Tony,Crack Peter J.,Duce James A.

Publisher

Elsevier BV

Subject

Physiology (medical),Biochemistry

Reference57 articles.

1. The impact of traumatic brain injuries: a global perspective;Hyder;NeuroRehabilitation,2007

2. Pathophysiology of cerebral ischemia and brain trauma: similarities and differences;Bramlett;J. Cereb. Blood Flow Metab.,2004

3. Neural injury following stroke: are Toll-like receptors the link between the immune system and the CNS?;Downes;Br. J. Pharmacol,2010

4. Temporal changes in free iron levels after brain ischemia: relevance to the timing of iron chelation therapy in stroke;Millerot-Serrurot;Neurochem. Int.,2008

5. MRI detection of secondary damage after stroke: chronic iron accumulation in the thalamus of the rat brain;Justicia;Stroke,2008

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