The transcriptional response to oxidative stress is part of, but not sufficient for, insulin resistance in adipocytes-Reference-Cited by-同舟云学术

The transcriptional response to oxidative stress is part of, but not sufficient for, insulin resistance in adipocytes

Published:2018-01-29 Issue:1 Volume:8 Page:
ISSN:2045-2322
Container-title:Scientific Reports
language:en
Short-container-title:Sci Rep

Author:

Chaudhuri Rima,Krycer James R.,Fazakerley Daniel J.^ORCID,Fisher-Wellman Kelsey H.,Su Zhiduan,Hoehn Kyle L.^ORCID,Yang Jean Yee Hwa,Kuncic Zdenka^ORCID,Vafaee Fatemeh^ORCID,James David E.

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/s41598-018-20104-x.pdf

Reference65 articles.

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2. Paglialunga, S., Ludzki, A., Root-McCaig, J. & Holloway, G. P. In adipose tissue, increased mitochondrial emission of reactive oxygen species is important for short-term high-fat diet-induced insulin resistance in mice. Diabetologia 58, 1071–1080, https://doi.org/10.1007/s00125-015-3531-x (2015).

3. Hoehn, K. L. et al. Insulin resistance is a cellular antioxidant defense mechanism. Proc Natl Acad Sci USA 106, 17787–17792, https://doi.org/10.1073/pnas.0902380106 (2009).

4. Furukawa, S. et al. Increased oxidative stress in obesity and its impact on metabolic syndrome. J Clin Invest 114, 1752–1761, https://doi.org/10.1172/JCI21625 (2004).

5. Houstis, N., Rosen, E. D. & Lander, E. S. Reactive oxygen species have a causal role in multiple forms of insulin resistance. Nature 440, 944–948, https://doi.org/10.1038/nature04634 (2006).

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