Diet induced obesity is independent of metabolic endotoxemia and TLR4 signalling, but markedly increases hypothalamic expression of the acute phase protein, SerpinA3N-Reference-Cited by-同舟云学术

Diet induced obesity is independent of metabolic endotoxemia and TLR4 signalling, but markedly increases hypothalamic expression of the acute phase protein, SerpinA3N

Published:2018-10-23 Issue:1 Volume:8 Page:
ISSN:2045-2322
Container-title:Scientific Reports
language:en
Short-container-title:Sci Rep

Author:

Dalby Matthew J.^ORCID,Aviello Gabriella,Ross Alexander W.,Walker Alan W.^ORCID,Barrett Perry,Morgan Peter J.^ORCID

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/s41598-018-33928-4.pdf

Reference66 articles.

1. Williams, L. M. et al. The development of diet-induced obesity and glucose intolerance in C57BL/6 mice on a high-fat diet consists of distinct phases. PloS one 9, e106159 (2014).

2. De Souza, C. T. et al. Consumption of a fat-rich diet activates a proinflammatory response and induces insulin resistance in the hypothalamus. Endocrinology 146, 4192–4199 (2005).

3. Thaler, J. P. et al. Obesity is associated with hypothalamic injury in rodents and humans. Journal of Clinical Investigation 122, 153–162 (2012).

4. Wisse, B. E. & Schwartz, M. W. Does hypothalamic inflammation cause obesity? Cell Metabolism 10, 241–242 (2009).

5. Cani, P. D. et al. Metabolic endotoxemia initiates obesity and insulin resistance. Diabetes 56, 1761–1772 (2007).

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