A critical role for p38MAPK signalling pathway during reprogramming of human fibroblasts to iPSCs
Author:
Publisher
Springer Science and Business Media LLC
Subject
Multidisciplinary
Link
http://www.nature.com/articles/srep41693.pdf
Reference52 articles.
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2. Allen, M. et al. Deficiency of the stress kinase p38alpha results in embryonic lethality: characterization of the kinase dependence of stress responses of enzyme-deficient embryonic stem cells. The Journal of experimental medicine 191, 859–870 (2000).
3. Adams, R. H. et al. Essential role of p38alpha MAP kinase in placental but not embryonic cardiovascular development. Molecular cell 6, 109–116 (2000).
4. Chakraborty, S., Kang, B., Huang, F. & Guo, Y. L. Mouse embryonic stem cells lacking p38alpha and p38delta can differentiate to endothelial cells, smooth muscle cells, and epithelial cells. Differentiation; research in biological diversity 78, 143–150, doi: 10.1016/j.diff.2009.05.006 (2009).
5. Aouadi, M. et al. p38 mitogen-activated protein kinase activity commits embryonic stem cells to either neurogenesis or cardiomyogenesis. Stem Cells 24, 1399–1406, doi: 10.1634/stemcells.2005-0398 (2006).
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