Repurposing drugs: Ca2+ ion dependency that can be exploited to treat keratitis caused by Acanthamoeba castellanii
Author:
Publisher
Springer Science and Business Media LLC
Subject
Ophthalmology
Link
http://www.nature.com/articles/s41433-019-0597-6.pdf
Reference6 articles.
1. Carnt N, Hoffman JJ, Verma S, et al. Acanthamoeba keratitis: confirmation of the UK outbreak and a prospective case-control study identifying contributing risk factors. Br J Ophthalmol. 2018. https://doi.org/10.1136/bjophthalmol-2018-312544 .
2. Trocha LK, Stobienia O. Response of Acanthamoeba castellanii mitochondria to oxidative stress. Acta Biochim Pol. 2007;54:797–803.
3. Baig AM, Rana Z, Waliani N, Karim S, Rajabali M. Evidence of human-like Ca2+ channels and effects of Ca2+ channel blockers in Acanthamoeba castellanii. Chem Biol Drug Des. 2019;93:351–63. https://doi.org/10.1111/cbdd.13421 .
4. Baig AM, Rana Z, Mannan M, Tariq S, Ahmad HR. Antibiotic effects of loperamide: homology of human targets of loperamide with targets in Acanthamoeba spp. Recent Pat Antiinfect Drug Discov. 2017;12:44–60. https://doi.org/10.2174/1574891X12666170425170544 .
5. Najjar DM, Cohen EJ, Rapuano CJ, Laibson PR. EDTA chelation for calcific band keratopathy: results and long-term follow-up. Am J Ophthalmol. 2004;137:1056–64. PubMed PMID: 15183790
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