STAT5 programs a distinct subset of GM-CSF-producing T helper cells that is essential for autoimmune neuroinflammation-Reference-Cited by-同舟云学术

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STAT5 programs a distinct subset of GM-CSF-producing T helper cells that is essential for autoimmune neuroinflammation

Published:2014-11-21 Issue:12 Volume:24 Page:1387-1402
ISSN:1001-0602
Container-title:Cell Research
language:en
Short-container-title:Cell Res

Author:

Sheng Wanqiang,Yang Fan,Zhou Yi,Yang Henry,Low Pey Yng,Kemeny David Michael,Tan Patrick,Moh Akira,Kaplan Mark H,Zhang Yongliang,Fu Xin-Yuan

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology,Molecular Biology

Link

http://www.nature.com/articles/cr2014154.pdf

Reference54 articles.

1. O'Shea JJ, Paul WE . Mechanisms underlying lineage commitment and plasticity of helper CD4+ T cells. Science 2010; 327:1098–1102.

2. Littman DR, Rudensky AY . TH17 and regulatory T cells in mediating and restraining inflammation. Cell 2010; 140:845–858.

3. Dong C . TH17 cells in development: an updated view of their molecular identity and genetic programming. Nat Rev Immunol 2008; 8:337–348.

4. Kaplan MH, Sun YL, Hoey T, Grusby MJ . Impaired IL-12 responses and enhanced development of Th2 cells in Stat4-deficient mice. Nature 1996; 382:174–177.

5. Thierfelder WE, van Deursen JM, Yamamoto K, et al. Requirement for Stat4 in interleukin-12-mediated responses of natural killer and T cells. Nature 1996; 382:171–174.

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