Transcription factor IRF8 directs a silencing programme for TH17 cell differentiation-Reference-Cited by-同舟云学术

Transcription factor IRF8 directs a silencing programme for TH17 cell differentiation

Published:2011-05-17 Issue:1 Volume:2 Page:
ISSN:2041-1723
Container-title:Nature Communications
language:en
Short-container-title:Nat Commun

Author:

Ouyang Xinshou,Zhang Ruihua,Yang Jianjun,Li Qingshan,Qin Lihui,Zhu Chen,Liu Jianguo,Ning Huan,Shin Min Sun,Gupta Monica,Qi Chen-Feng,He John Cijiang,Lira Sergio A.,Morse Herbert C.,Ozato Keiko,Mayer Lloyd,Xiong Huabao

Publisher

Springer Science and Business Media LLC

Subject

General Physics and Astronomy,General Biochemistry, Genetics and Molecular Biology,General Chemistry

Link

http://www.nature.com/articles/ncomms1311.pdf

Reference50 articles.

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2. Weaver, C. T., Harrington, L. E., Mangan, P. R., Gavrieli, M. & Murphy, K. M. Th17: an effector CD4 T cell lineage with regulatory T cell ties. Immunity 24, 677–688 (2006).

3. Zheng, Y. et al. Interleukin-22, a T(H)17 cytokine, mediates IL-23-induced dermal inflammation and acanthosis. Nature 445, 648–651 (2007).

4. Liang, S. C. et al. Interleukin (IL)-22 and IL-17 are coexpressed by Th17 cells and cooperatively enhance expression of antimicrobial peptides. J. Exp. Med. 203, 2271–2279 (2006).

5. Veldhoen, M., Hocking, R. J., Atkins, C. J., Locksley, R. M. & Stockinger, B. TGFbeta in the context of an inflammatory cytokine milieu supports de novo differentiation of IL-17-producing T cells. Immunity 24, 179–189 (2006).

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