A systemic view of Alzheimer disease — insights from amyloid-β metabolism beyond the brain-Reference-Cited by-同舟云学术

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A systemic view of Alzheimer disease — insights from amyloid-β metabolism beyond the brain

Published:2017-09-29 Issue:10 Volume:13 Page:612-623
ISSN:1759-4758
Container-title:Nature Reviews Neurology
language:en
Short-container-title:Nat Rev Neurol

Author:

Wang Jun,Gu Ben J.,Masters Colin L.,Wang Yan-Jiang

Publisher

Springer Science and Business Media LLC

Subject

Cellular and Molecular Neuroscience,Neurology (clinical)

Link

http://www.nature.com/articles/nrneurol.2017.111.pdf

Reference175 articles.

1. Mangialasche, F., Solomon, A., Winblad, B., Mecocci, P. & Kivipelto, M. Alzheimer's disease: clinical trials and drug development. Lancet Neurol. 9, 702–716 (2010).

2. Berk, C., Paul, G. & Sabbagh, M. Investigational drugs in Alzheimer's disease: current progress. Expert Opin. Investig. Drugs 23, 837–846 (2014).

3. Selkoe, D. J. & Hardy, J. The amyloid hypothesis of Alzheimer's disease at 25 years. EMBO Mol. Med. 8, 595–608 (2016). This article reviews new evidence supporting the concept that an imbalance between production and clearance of Aβ is a very early, often initiating factor, in AD — a widely debated issue.

4. Yankner, B. A. & Mesulam, M. M. Seminars in medicine of the Beth Israel Hospital, Boston. β-Amyloid and the pathogenesis of Alzheimer's disease. N. Engl. J. Med. 325, 1849–1857 (1991).

5. Roher, A. E. et al. Amyloid β peptides in human plasma and tissues and their significance for Alzheimer's disease. Alzheimers Dement. 5, 18–29 (2009). This study evaluates Aβ levels in brain, peripheral organs and tissues, suggesting that brain as well as plasma Aβ levels are the consequence of intricate relationships between central and peripehral sources.

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