Total insulin and IGF-I resistance in pancreatic β cells causes overt diabetes-Reference-Cited by-同舟云学术

Total insulin and IGF-I resistance in pancreatic β cells causes overt diabetes

Published:2006-04-23 Issue:5 Volume:38 Page:583-588
ISSN:1061-4036
Container-title:Nature Genetics
language:en
Short-container-title:Nat Genet

Author:

Ueki Kohjiro,Okada Terumasa,Hu Jiang,Liew Chong Wee,Assmann Anke,Dahlgren Gabriella M,Peters Jennifer L,Shackman Jonathan G,Zhang Min,Artner Isabella,Satin Leslie S,Stein Roland,Holzenberger Martin,Kennedy Robert T,Kahn C Ronald,Kulkarni Rohit N

Publisher

Springer Science and Business Media LLC

Subject

Genetics

Link

http://www.nature.com/articles/ng1787.pdf

Reference30 articles.

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3. Kulkarni, R.N. et al. Tissue-specific knockout of the insulin receptor in pancreatic β cells creates an insulin secretory defect similar to that in Type 2 diabetes. Cell 96, 329–339 (1999).

4. Kulkarni, R.N. et al. beta-cell-specific deletion of the Igf1 receptor leads to hyperinsulinemia and glucose intolerance but does not alter beta-cell mass. Nat. Genet. 31, 111–115 (2002).

5. Otani, K. et al. Reduced beta-cell mass and altered glucose sensing impair insulin-secretory function in betaIRKO mice. Am. J. Physiol. Endocrinol. Metab. 286, E41–E49 (2004).

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