SRSF2 mutations drive oncogenesis by activating a global program of aberrant alternative splicing in hematopoietic cells-Reference-Cited by-同舟云学术

SRSF2 mutations drive oncogenesis by activating a global program of aberrant alternative splicing in hematopoietic cells

Published:2018-06-01 Issue:12 Volume:32 Page:2659-2671
ISSN:0887-6924
Container-title:Leukemia
language:en
Short-container-title:Leukemia

Author:

Liang Yang,Tebaldi Toma,Rejeski Kai,Joshi Poorval,Stefani Giovanni,Taylor Ashley,Song Yuanbin,Vasic Radovan,Maziarz Jamie,Balasubramanian Kunthavai,Ardasheva Anastasia,Ding Alicia,Quattrone Alessandro,Halene Stephanie^ORCID

Publisher

Springer Science and Business Media LLC

Subject

Oncology,Cancer Research,Hematology

Link

http://www.nature.com/articles/s41375-018-0152-7.pdf

Reference56 articles.

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2. Meggendorfer M, Roller A, Haferlach T, Eder C, Dicker F, Grossmann V, et al. SRSF2 mutations in 275 cases with chronic myelomonocytic leukemia (CMML). Blood. 2012;120:3080–8.

3. Papaemmanuil E, Gerstung M, Malcovati L, Tauro S, Gundem G, Van Loo P, et al. Clinical and biological implications of driver mutations in myelodysplastic syndromes. Blood. 2013;122:3616–27.

4. Lee SC-W, Dvinge H, Kim E, Cho H, Micol J-B, Chung YR, et al. Modulation of splicing catalysis for therapeutic targeting of leukemia with mutations in genes encoding spliceosomal proteins. Nat Med. 2016;22:672–8.

5. Howard JM, Sanford JR. The RNAissance family: SR proteins as multifaceted regulators of gene expression. Wiley Interdiscip Rev RNA. 2015;6:93–110.

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