HDAC7 is a major contributor in the pathogenesis of infant t(4;11) proB acute lymphoblastic leukemia-Reference-Cited by-同舟云学术

HDAC7 is a major contributor in the pathogenesis of infant t(4;11) proB acute lymphoblastic leukemia

Published:2020-12-01 Issue:7 Volume:35 Page:2086-2091
ISSN:0887-6924
Container-title:Leukemia
language:en
Short-container-title:Leukemia

Author:

de Barrios Oriol,Galaras Alexandros,Trincado Juan L.^ORCID,Azagra Alba^ORCID,Collazo Olga,Meler Ainara^ORCID,Agraz-Doblas Antonio,Bueno Clara,Ballerini Paola,Cazzaniga Giovanni,Stam Ronald W.^ORCID,Varela Ignacio^ORCID,De Lorenzo Paola,Valsecchi Maria Grazia,Hatzis Pantelis^ORCID,Menéndez Pablo,Parra Maribel^ORCID

Publisher

Springer Science and Business Media LLC

Subject

Oncology,Cancer Research,Hematology

Link

http://www.nature.com/articles/s41375-020-01097-x.pdf

Reference16 articles.

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2. Bueno C, Montes R, Catalina P, Rodríguez R, Menendez P. Insights into the cellular origin and etiology of the infant pro-B acute lymphoblastic leukemia with MLL-AF4 rearrangement. Leukemia. 2011;25:400–10.

3. Meyer C, Burmeister T, Gröger D, Tsaur G, Fechina L, Renneville A, et al. The MLL recombinome of acute leukemias in 2017. Leukemia. 2018;32:273–84.

4. Kowarz E, Burmeister T, Lo Nigro L, Jansen MWJC, Delabesse E, Klingebiel T, et al. Complex MLL rearrangements in t(4;11) leukemia patients with absent AF4·MLL fusion allele. Leukemia. 2007;21:1232–8.

5. Pieters R, Schrappe M, De Lorenzo P, Hann I, De Rossi G, Felice M, et al. A treatment protocol for infants younger than 1 year with acute lymphoblastic leukemia (Interfant-99): an observational study and a multicentre randomized trial. Lancet. 2007;370:240–50.

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