Insights into the cellular origin and etiology of the infant pro-B acute lymphoblastic leukemia with MLL-AF4 rearrangement
Author:
Publisher
Springer Science and Business Media LLC
Subject
Oncology,Cancer Research,Hematology
Link
http://www.nature.com/articles/leu2010284.pdf
Reference98 articles.
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2. Eguchi M, Eguchi-Ishimae M, Knight D, Kearney L, Slany R, Greaves M . MLL chimeric protein activation renders cells vulnerable to chromosomal damage: an explanation for the very short latency of infant leukemia. Genes Chromosomes Cancer 2006; 45: 754–760.
3. Eguchi M, Eguchi-Ishimae M, Greaves M . Molecular pathogenesis of MLL-associated leukemias. Int J Hematol 2005; 82: 9–20.
4. Krivtsov AV, Armstrong SA . MLL translocations, histone modifications and leukaemia stem-cell development. Nat Rev Cancer 2007; 7: 823–833.
5. Pui CH . Acute lymphoblastic leukemia in children. Curr Opin Oncol 2000; 12: 3–12.
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