Reply to: ‘Interaction between RAAS inhibitors and ACE2 in the context of COVID-19’
Author:
Publisher
Springer Science and Business Media LLC
Subject
Cardiology and Cardiovascular Medicine
Link
http://www.nature.com/articles/s41569-020-0369-9.pdf
Reference10 articles.
1. Mourad, J.-J. & Levy, B. I. Interaction between RAAS inhibitors and ACE2 in the context of COVID-19. Nat. Rev. Cardiol. https://doi.org/10.1038/s41569-020-0368-x (2020).
2. Zheng, Y. Y., Ma, Y. T., Zhang, J. Y. & Xie, X. COVID-19 and the cardiovascular system. Nat. Rev. Cardiol. https://doi.org/10.1038/s41569-020-0360-5 (2020).
3. Ferrario, C. M. et al. Effect of angiotensin-converting enzyme inhibition and angiotensin II receptor blockers on cardiac angiotensin-converting enzyme 2. Circulation 111, 2605–2610 (2005).
4. Li, Y. et al. Angiotensin-converting enzyme inhibition attenuates lipopolysaccharide-induced lung injury by regulating the balance between angiotensin-converting enzyme and angiotensin-converting enzyme 2 and inhibiting mitogen-activated protein kinase activation. Shock 43, 395–404 (2015).
5. Wösten-van Asperen, R. M. et al. Acute respiratory distress syndrome leads to reduced ratio of ACE/ACE2 activities and is prevented by angiotensin-(1–7) or an angiotensin II receptor antagonist. J. Pathol. 225, 618–627 (2011).
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