Suppressing aberrant GluN3A expression rescues synaptic and behavioral impairments in Huntington's disease models-Reference-Cited by-同舟云学术

Suppressing aberrant GluN3A expression rescues synaptic and behavioral impairments in Huntington's disease models

Published:2013-07-14 Issue:8 Volume:19 Page:1030-1038
ISSN:1078-8956
Container-title:Nature Medicine
language:en
Short-container-title:Nat Med

Author:

Marco Sonia,Giralt Albert,Petrovic Milos M,Pouladi Mahmoud A,Martínez-Turrillas Rebeca,Martínez-Hernández José,Kaltenbach Linda S,Torres-Peraza Jesús,Graham Rona K,Watanabe Masahiko,Luján Rafael,Nakanishi Nobuki,Lipton Stuart A,Lo Donald C,Hayden Michael R,Alberch Jordi,Wesseling John F,Pérez-Otaño Isabel

Publisher

Springer Science and Business Media LLC

Subject

General Biochemistry, Genetics and Molecular Biology,General Medicine

Link

http://www.nature.com/articles/nm.3246.pdf

Reference59 articles.

1. DiFiglia, M. et al. Aggregation of huntingtin in neuronal intranuclear inclusions and dystrophic neurites in brain. Science 277, 1990–1993 (1997).

2. DiFiglia, M. et al. Huntingtin is a cytoplasmic protein associated with vesicles in human and rat brain neurons. Neuron 14, 1075–1081 (1995).

3. Schaffar, G. et al. Cellular toxicity of polyglutamine expansion proteins: mechanism of transcription factor deactivation. Mol. Cell 15, 95–105 (2004).

4. Kaltenbach, L.S. et al. Huntingtin interacting proteins are genetic modifiers of neurodegeneration. PLoS Genet. 3, e82 (2007).

5. Goehler, H. et al. A protein interaction network links GIT1, an enhancer of huntingtin aggregation, to Huntington's disease. Mol. Cell 15, 853–865 (2004).

Cited by 93 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Excitatory GluN1/GluN3A glycine receptors (eGlyRs) in brain signaling;Trends in Neurosciences;2023-08

2. A Neuroligin-1 mutation associated with Alzheimer’s disease produces memory and age-dependent impairments in hippocampal plasticity;iScience;2023-06

3. Activation of excitatory glycine NMDA receptors: At the mercy of a whimsical GluN1 subunit;Journal of General Physiology;2023-05-03

4. Allosteric modulation of GluN1/GluN3 NMDA receptors by GluN1-selective competitive antagonists;Journal of General Physiology;2023-04-20

5. Early TNF-Dependent Regulation of Excitatory and Inhibitory Synapses on Striatal Direct Pathway Medium Spiny Neurons in the YAC128 Mouse Model of Huntington's Disease;The Journal of Neuroscience;2022-12-14