Distribution of genomic breakpoints in chronic myeloid leukemia: analysis of 308 patients-Reference-Cited by-同舟云学术

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Distribution of genomic breakpoints in chronic myeloid leukemia: analysis of 308 patients

Published:2013-04-16 Issue:10 Volume:27 Page:2105-2107
ISSN:0887-6924
Container-title:Leukemia
language:en
Short-container-title:Leukemia

Author:

Ross D M,O'Hely M,Bartley P A,Dang P,Score J,Goyne J M,Sobrinho-Simoes M,Cross N C P,Melo J V,Speed T P,Hughes T P,Morley A A

Publisher

Springer Science and Business Media LLC

Subject

Oncology,Cancer Research,Hematology

Link

http://www.nature.com/articles/leu2013116.pdf

Reference15 articles.

1. Gillert E, Leis T, Repp R, Reichel M, Hosch A, Breitenlohner I et al. A DNA damage repair mechanism is involved in the origin of chromosomal translocations t(4;11) in primary leukemic cells. Oncogene 1999; 18: 4663–4671.

2. Reiter A, Saussele S, Grimwade D, Wiemels JL, Segal MR, Lafage-Pochitaloff M et al. Genomic anatomy of the specific reciprocal translocation t(15;17) in acute promyelocytic leukemia. Genes Chromosomes Cancer 2003; 36: 175–188.

3. Zhang JG, Goldman JM, Cross NC . Characterization of genomic BCR-ABL breakpoints in chronic myeloid leukaemia by PCR. Br J Haematol 1995; 90: 138–146.

4. Mattarucchi E, Guerini V, Rambaldi A, Campiotti L, Venco A, Pasquali F et al. Microhomologies and interspersed repeat elements at genomic breakpoints in chronic myeloid leukemia. Genes Chromosomes Cancer 2008; 47: 625–632.

5. Jeffs AR, Benjes SM, Smith TL, Sowerby SJ, Morris CM . The BCR gene recombines preferentially with Alu elements in complex BCR-ABL translocations of chronic myeloid leukaemia. Hum Mol Genet 1998; 7: 767–776.

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