SYK regulates mTOR signaling in AML-Reference-Cited by-同舟云学术

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SYK regulates mTOR signaling in AML

Published:2013-03-28 Issue:11 Volume:27 Page:2118-2128
ISSN:0887-6924
Container-title:Leukemia
language:en
Short-container-title:Leukemia

Author:

Carnevale J,Ross L,Puissant A,Banerji V,Stone R M,DeAngelo D J,Ross K N,Stegmaier K

Publisher

Springer Science and Business Media LLC

Subject

Oncology,Cancer Research,Hematology

Link

http://www.nature.com/articles/leu201389.pdf

Reference65 articles.

1. Juliusson G, Antunovic P, Derolf A, Lehmann S, Mollgard L, Stockelberg D et al. Age and acute myeloid leukemia: real world data on decision to treat and outcomes from the Swedish Acute Leukemia Registry. Blood 2009; 113: 4179–4187.

2. Castaigne S, Chomienne C, Daniel MT, Ballerini P, Berger R, Fenaux P et al. All-trans retinoic acid as a differentiation therapy for acute promyelocytic leukemia. I. Clinical results. Blood 1990; 76: 1704–1709.

3. Hahn CK, Berchuck JE, Ross KN, Kakoza RM, Clauser K, Schinzel AC et al. Proteomic and genetic approaches identify Syk as an AML target. Cancer Cell 2009; 16: 281–294.

4. Stegmaier K, Corsello SM, Ross KN, Wong JS, Deangelo DJ, Golub TR . Gefitinib induces myeloid differentiation of acute myeloid leukemia. Blood 2005; 106: 2841–2848.

5. Stegmaier K, Ross KN, Colavito SA, O'Malley S, Stockwell BR, Golub TR . Gene expression-based high-throughput screening(GE-HTS) and application to leukemia differentiation. Nat Genet 2004; 36: 257–263.

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