Podocyte injury in diabetic nephropathy: implications of angiotensin II – dependent activation of TRPC channels
Author:
Publisher
Springer Science and Business Media LLC
Subject
Multidisciplinary
Link
http://www.nature.com/articles/srep17637.pdf
Reference56 articles.
1. Zhang, P. et al. Global healthcare expenditure on diabetes for 2010 and 2030. Diabetes Res. Clin. Pract. 87, 293–301 (2010).
2. Tervaert, T. W. et al. Pathologic classification of diabetic nephropathy. J. Am. Soc. Nephrol. 21, 556–563 (2010).
3. Pagtalunan, M. E. et al. Podocyte loss and progressive glomerular injury in type II diabetes. J. Clin. Invest. 99, 342–348 (1997).
4. Wolf, G., Chen, S. & Ziyadeh, F. N. From the periphery of the glomerular capillary wall toward the center of disease: podocyte injury comes of age in diabetic nephropathy. Diabetes 54, 1626–1634 (2005).
5. Fogo, A. B. The targeted podocyte. J. Clin. Invest. 121, 2142–2145 (2011).
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