BCAS2 is involved in alternative mRNA splicing in spermatogonia and the transition to meiosis-Reference-Cited by-同舟云学术

BCAS2 is involved in alternative mRNA splicing in spermatogonia and the transition to meiosis

Published:2017-01-27 Issue:1 Volume:8 Page:
ISSN:2041-1723
Container-title:Nature Communications
language:en
Short-container-title:Nat Commun

Author:

Liu Wenbo,Wang Fengchao,Xu Qianhua,Shi Junchao,Zhang Xiaoxin,Lu Xukun,Zhao Zhen-Ao,Gao Zheng,Ma Huaixiao,Duan Enkui,Gao Fei,Gao Shaorong,Yi Zhaohong,Li Lei^ORCID

Publisher

Springer Science and Business Media LLC

Subject

General Physics and Astronomy,General Biochemistry, Genetics and Molecular Biology,General Chemistry

Link

http://www.nature.com/articles/ncomms14182.pdf

Reference64 articles.

1. Cieply, B. & Carstens, R. P. Functional roles of alternative splicing factors in human disease. Wires RNA 6, 311–326 (2015).

2. Calarco, J. A., Zhen, M. & Blencowe, B. J. Networking in a global world: Establishing functional connections between neural splicing regulators and their target transcripts. RNA 17, 775–791 (2011).

3. Kalsotra, A. & Cooper, T. A. Functional consequences of developmentally regulated alternative splicing. Nat. Rev. Genet. 12, 715–729 (2011).

4. Barbosa-Morais, N. L. et al. The evolutionary landscape of alternative splicing in vertebrate species. Science 338, 1587–1593 (2012).

5. Nilsen, T. W. & Graveley, B. R. Expansion of the eukaryotic proteome by alternative splicing. Nature 463, 457–463 (2010).

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