Disruption of the FasL/Fas axis protects against inflammation-derived tumorigenesis in chronic liver disease-Reference-Cited by-同舟云学术

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Disruption of the FasL/Fas axis protects against inflammation-derived tumorigenesis in chronic liver disease

Published:2019-02 Issue:2 Volume:10 Page:
ISSN:2041-4889
Container-title:Cell Death & Disease
language:en
Short-container-title:Cell Death Dis

Author:

Cubero Francisco Javier,Woitok Marius Maximilian,Zoubek Miguel E.,de Bruin Alain,Hatting Maximilian,Trautwein Christian

Funder

Consejería de Sanidad, Comunidad de Madrid

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Cell Biology,Cellular and Molecular Neuroscience,Immunology

Link

http://www.nature.com/articles/s41419-019-1391-x.pdf

Reference27 articles.

1. Fecho, K. & Cohen, P. L. Fas ligand (gld)- and Fas (lpr)-deficient mice do not show alterations in the extravasation or apoptosis of inflammatory neutrophils. J. Leukoc. Biol. 64, 373–383 (1998).

2. Faubion, W. A. & Gores, G. J. Death receptors in liver biology and pathobiology. Hepatology 29, 1–4 (1999).

3. Ito, Y. et al. Fas antigen expression in hepatocellular carcinoma tissues. Oncol. Rep. 5, 41–44 (1998).

4. Abrams, S. I. Positive and negative consequences of Fas/Fas ligand interactions in the antitumor response. Front. Biosci. 10, 809–821 (2005).

5. Nicholes, K. et al. A mouse model of hepatocellular carcinoma: ectopic expression of fibroblast growth factor 19 in skeletal muscle of transgenic mice. Am. J. Pathol. 160, 2295–2307 (2002).

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