Apelin inhibited epithelial−mesenchymal transition of podocytes in diabetic mice through downregulating immunoproteasome subunits β5i
Author:
Publisher
Springer Science and Business Media LLC
Subject
Cancer Research,Cell Biology,Cellular and Molecular Neuroscience,Immunology
Link
http://www.nature.com/articles/s41419-018-1098-4.pdf
Reference25 articles.
1. Loeffler, I., Liebisch, M. & Wolf, G. Collagen VIII influences epithelial phenotypic changes in experimental diabetic nephropathy. Am. J. Physiol. Ren. Physiol. 303, F733–F745 (2012).
2. Loeffler, I. & Wolf, G. Epithelial-to-mesenchymal transition in diabetic nephropathy: fact or fiction? Cells 4, 631–652 (2015).
3. Hu, C. et al. Insights into the mechanisms involved in the expression and regulation of extracellular matrix proteins in diabetic nephropathy. Curr. Med. Chem. 22, 2858–2870 (2015).
4. Ziyadeh, F. N. & Wolf, G. Pathogenesis of the podocytopathy and proteinuria in diabetic glomerulopathy. Curr. Diabetes Rev. 4, 39–45 (2008).
5. Kocer, D., Karakukcu, C., Ozturk, F., Eroglu, E. & Kocyigit, I. Evaluation of fibrosis markers: apelin and transforming growth factor-beta1 in autosomal dominant polycystic kidney disease patients. Ther. Apher. Dial. 20, 517–522 (2016).
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