STK17B promotes carcinogenesis and metastasis via AKT/GSK-3β/Snail signaling in hepatocellular carcinoma-Reference-Cited by-同舟云学术

STK17B promotes carcinogenesis and metastasis via AKT/GSK-3β/Snail signaling in hepatocellular carcinoma

Published:2018-02 Issue:2 Volume:9 Page:
ISSN:2041-4889
Container-title:Cell Death & Disease
language:en
Short-container-title:Cell Death Dis

Author:

Lan Yaliang,Han Jihua,Wang Yan,Wang Jiabei,Yang Guangchao,Li Keyu,Song Ruipeng,Zheng Tongsen,Liang Yingjian,Pan Shangha,Liu Xirui,Zhu Mingxi,Liu Yao,Meng Fanzheng,Mohsin Manzoor,Cui Yifeng,Zhang Bo,Subash Sharma,Liu Lianxin

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Cell Biology,Cellular and Molecular Neuroscience,Immunology

Link

http://www.nature.com/articles/s41419-018-0262-1.pdf

Reference35 articles.

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3. Sanjo, H., Kawai, T. & Akira, S. DRAKs, novel serine/threonine kinases related to death-associated protein kinase that trigger apoptosis. J. Biol. Chem. 273, 29066–29071 (1998).

4. McGargill, M. A., Wen, B. G., Walsh, C. & MHedrick, S. M. A deficiency in Drak2 results in a T cell hypersensitivity and an unexpected resistance to autoimmunity. Immunity 21, 781–791 (2004).

5. Edwards, B. A. et al. Drak2 is not required for tumor surveillance and suppression. Int Immunol. 27, 161–166 (2015).

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