Drak2 is not required for tumor surveillance and suppression-Reference-Cited by-同舟云学术

Drak2 is not required for tumor surveillance and suppression

Published:2015-01-07 Issue:3 Volume:27 Page:161-166
ISSN:1460-2377
Container-title:International Immunology
language:en
Short-container-title:

Author:

Edwards Benjamin A.,Harris Tarsha L.,Floersh Helen,Lukens John R.,Zaki Md. Hasan,Vogel Peter,Kanneganti Thirumala-Devi,Bui Jack D.,McGargill Maureen A.

Publisher

Oxford University Press (OUP)

Subject

Immunology,General Medicine,Immunology and Allergy

Link

http://academic.oup.com/intimm/article-pdf/27/3/161/18323646/dxu146.pdf

Reference22 articles.

1. A deficiency in Drak2 results in a T cell hypersensitivity and an unexpected resistance to autoimmunity;McGargill;Immunity,2004

2. DRAK2, a lymphoid-enriched DAP kinase, regulates the TCR activation threshold during thymocyte selection;Friedrich;Int. Immunol,2005

3. DRAKs, novel serine/threonine kinases related to death-associated protein kinase that trigger apoptosis;Sanjo;J. Biol. Chem,1998

4. Drak2 regulates the survival of activated T cells and is required for organ-specific autoimmune disease;McGargill;J. Immunol,2008

5. Loss of DRAK2 signaling enhances allogeneic transplant survival by limiting effector and memory T cell responses;Weist;Am. J. Transplant,2012

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