Cellular energy stress induces AMPK-mediated regulation of glioblastoma cell proliferation by PIKE-A phosphorylation
Author:
Publisher
Springer Science and Business Media LLC
Subject
Cancer Research,Cell Biology,Cellular and Molecular Neuroscience,Immunology
Link
http://www.nature.com/articles/s41419-019-1452-1.pdf
Reference30 articles.
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2. Hu, Y., Liu, Z. & Ye, K. Phosphoinositol lipids bind to phosphatidylinositol 3 (PI3)-kinase enhancer GTPase and mediate its stimulatory effect on PI3-kinase and Akt signalings. Proc. Natl. Acad. Sci. USA 102, 16853–16858 (2005).
3. Qi, Q. & Ye, K. The roles of PIKE in tumorigenesis. Acta Pharmacol. Sin. 34, 991–997 (2013).
4. Ye, K. et al. Pike. A nuclear gtpase that enhances PI3kinase activity and is regulated by protein 4.1N. Cell 103, 919–930 (2000).
5. Cai, Y. et al. GGAP2/PIKE-a directly activates both the Akt and nuclear factor-kappaB pathways and promotes prostate cancer progression. Cancer Res. 69, 819–827 (2009).
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