miR‐31 modulates dystrophin expression: new implications for Duchenne muscular dystrophy therapy
Author:
Affiliation:
1. Department of Biology and Biotechnology ‘C. Darwin’, Institut Pasteur Cenci‐Bolognetti and IBPM—Sapienza, University of Rome Piazzele Aldo Moro 5 Rome 00185 Italy
Publisher
EMBO
Subject
Genetics,Molecular Biology,Biochemistry
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1038/embor.2010.208
Reference24 articles.
1. Therapeutic antisense-induced exon skipping in cultured muscle cells from six different DMD patients
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3. Targeted Exon Skipping in Transgenic hDMD Mice: A Model for Direct Preclinical Screening of Human-Specific Antisense Oligonucleotides
4. MicroRNAs Involved in Molecular Circuitries Relevant for the Duchenne Muscular Dystrophy Pathogenesis Are Controlled by the Dystrophin/nNOS Pathway
5. The role of microRNA-1 and microRNA-133 in skeletal muscle proliferation and differentiation
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