AML1/ETO sensitizes via TRAIL acute myeloid leukemia cells to the pro-apoptotic effects of hypoxia-Reference-Cited by-同舟云学术

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AML1/ETO sensitizes via TRAIL acute myeloid leukemia cells to the pro-apoptotic effects of hypoxia

Published:2013-03 Issue:3 Volume:4 Page:e536-e536
ISSN:2041-4889
Container-title:Cell Death & Disease
language:en
Short-container-title:Cell Death Dis

Author:

Barbetti V,Tusa I,Cipolleschi M G,Rovida E,Dello Sbarba P

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Cell Biology,Cellular and Molecular Neuroscience,Immunology

Link

http://www.nature.com/articles/cddis201349.pdf

Reference35 articles.

1. Wang J, Hoshino T, Redner RL, Kajigaya S, Liu JM . Protein ETO, fusion partner in t(8;21) acute myeloid leukemia, represses transcription by interaction with the human N-CoR/mSin3/HDAC1complex. Proc Natl Acad Sci USA 1998; 95: 10860–10865.

2. Downing JR . The AML1-ETO chimaeric transcription factor in acute myeloid leukaemia: biology and clinical significance. Br J Haematol 1999; 106: 296–308.

3. Gardini A, Cesaroni M, Luzi L, Okumura AJ, Biggs JR, Minardi SP et al. M. AML1/ETO oncoprotein is directed to AML1 binding regions and co-localizes with AML1 and HEB on its targets. PLoS Genet 2008; 4: e1000275.

4. Licht JD . AML1 and the AML1-ETO fusion protein in the pathogenesis of t(8;21) AML. Oncogene 2001; 20: 5660–5679.

5. Cipolleschi MG, Dello Sbarba P, Olivotto M . The role of hypoxia in the maintenance of hematopoietic stem cells. Blood 1993; 82: 2031–2037.

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