TGF-β induces TIAF1 self-aggregation via type II receptor-independent signaling that leads to generation of amyloid β plaques in Alzheimer's disease-Reference-Cited by-同舟云学术

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TGF-β induces TIAF1 self-aggregation via type II receptor-independent signaling that leads to generation of amyloid β plaques in Alzheimer's disease

Published:2010-12 Issue:12 Volume:1 Page:e110-e110
ISSN:2041-4889
Container-title:Cell Death & Disease
language:en
Short-container-title:Cell Death Dis

Author:

Lee M-H,Lin S-R,Chang J-Y,Schultz L,Heath J,Hsu L-J,Kuo Y-M,Hong Q,Chiang M-F,Gong C-X,Sze C-I,Chang N-S

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Cell Biology,Cellular and Molecular Neuroscience,Immunology

Link

http://www.nature.com/articles/cddis201083.pdf

Reference39 articles.

1. Hanger DP, Anderton BH, Noble W . Tau phosphorylation: the therapeutic challenge for neurodegenerative disease. Trends Mol Med 2009; 15: 112–119.

2. Tesseur I, Zou K, Esposito L, Bard F, Berber E, Can JV et al. Deficiency in neuronal TGF-beta signaling promotes neurodegeneration and Alzheimer's pathology. J Clin Invest 2006; 116: 3060–3069.

3. Salins P, He Y, Olson K, Glazner G, Kashour T, Amara F . TGF-beta1 is increased in a transgenic mouse model of familial Alzheimer's disease and causes neuronal apoptosis. Neurosci Lett 2008; 430: 81–86.

4. Peila R, Yucesoy B, White LR, Johnson V, Kashon ML, Wu K et al. A TGF-beta1 polymorphism association with dementia and neuropathologies: the HAAS. Neurobiol Aging 2007; 28: 1367–1373.

5. Town T, Laouar Y, Pittenger C, Mori T, Szekely CA, Tan J et al. Blocking TGF-beta-Smad2/3 innate immune signaling mitigates Alzheimer-like pathology. Nat Med 2008; 14: 681–687.

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