Fructose drives mitochondrial metabolic reprogramming in podocytes via Hmgcs2-stimulated fatty acid degradation-Reference-Cited by-同舟云学术

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Fructose drives mitochondrial metabolic reprogramming in podocytes via Hmgcs2-stimulated fatty acid degradation

Published:2021-07-09 Issue:1 Volume:6 Page:
ISSN:2059-3635
Container-title:Signal Transduction and Targeted Therapy
language:en
Short-container-title:Sig Transduct Target Ther

Author:

Fang Lei,Li Tu-Shuai,Zhang Jing-Zi,Liu Zhi-Hong,Yang Jie,Wang Bing-Hao,Wang Yu-Meng,Zhou Jie,Kong Ling-Dong

Funder

National Natural Science Foundation of China

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Genetics

Link

http://www.nature.com/articles/s41392-021-00570-y.pdf

Reference4 articles.

1. Afshin, A. et al. Health effects of overweight and obesity in 195 countries over 25 years. N. Engl. J. Med. 377, 13–27 (2017).

2. Galvan, D. L. et al. Real-time in vivo mitochondrial redox assessment confirms enhanced mitochondrial reactive oxygen species in diabetic nephropathy. Kidney Int. 92, 1282–1287 (2017).

3. Qi, H. et al. Glomerular endothelial mitochondrial dysfunction is essential and characteristic of diabetic kidney disease susceptibility. Diabetes 66, 763–778 (2017).

4. Fan, C. Y. et al. Betaine supplementation protects against high-fructose-induced renal injury in rats. J. Nutr. Biochem. 25, 353–362 (2014).

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1. Loss of Nup155 promotes high fructose-driven podocyte senescence by inhibiting INO80 mRNA nuclear export;Journal of Advanced Research;2024-08

2. High expression of 3-hydroxy-3-methylglutaryl-CoA synthase 2 (HMGCS2) associated with Diquat-induced damage;Ecotoxicology and Environmental Safety;2024-08

3. Downregulation of HMGCS2 mediated AECIIs lipid metabolic alteration promotes pulmonary fibrosis by activating fibroblasts;Respiratory Research;2024-04-24

4. Small extracellular vesicles from young plasma reverse age-related functional declines by improving mitochondrial energy metabolism;Nature Aging;2024-04-16

5. Notoginsenoside Fc, a novel renoprotective agent, ameliorates glomerular endothelial cells pyroptosis and mitochondrial dysfunction in diabetic nephropathy through regulating HMGCS2 pathway;Phytomedicine;2024-04

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