Loss of Nup155 promotes high fructose-driven podocyte senescence by inhibiting INO80 mRNA nuclear export-Reference-Cited by-同舟云学术

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Loss of Nup155 promotes high fructose-driven podocyte senescence by inhibiting INO80 mRNA nuclear export

Published:2024-08 Issue: Volume: Page:
ISSN:2090-1232
Container-title:Journal of Advanced Research
language:en
Short-container-title:Journal of Advanced Research

Author:

Chen Li,Xu Tangdi,Wang Zixuan,Wang Chengzhi,Fang Lei,Kong Lingdong

Funder

National Key Research and Development Program of China Stem Cell and Translational Research

National Natural Science Foundation of China

National Key Research and Development Program of China

National Key Clinical Specialty Discipline Construction Program of China

National Aerospace Science Foundation of China

Publisher

Elsevier BV

Reference37 articles.

1. Podocyte injury and its consequences;Nagata;Kidney Int,2016

2. Reduction of anaerobic glycolysis contributes to angiotensin II-induced podocyte injury with foot process effacement;Chen;Kidney Int,2023

3. SSBP1 drives high fructose-induced glomerular podocyte ferroptosis via activating DNA-PK/p53 pathway;Wu;Redox Biol,2022

4. Manganese induces podocyte injury through regulating MTDH/ALKBH5/NLRP10 axis: Combined analysis at epidemiology and molecular biology levels;Liang;Environ Int,2024

5. Cellular senescence: defining a path forward;Gorgoulis;Cell,2019

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