Factors influencing the low-frequency associated nicotinic ACh autoreceptor-mediated depression of ACh release from rat motor nerve terminals

Author:

Prior Chris,Singh Shila

Publisher

Wiley

Subject

Pharmacology

Reference33 articles.

1. ω-Conotoxin does not block the verapamil-sensitive calcium channels at mouse motor nerve terminals;ANDERSON;Neurosci. Lett.,1987

2. Transversally cut diaphragm preparation from the rat;BARSTAD;Arch. Int. Pharmacodyn. Therap.,1968

3. Pharmacological characterization of recombinant human neuronal nicotinic acetylcholine receptors hα2β2, hα2β4, hα3β2, hα3β4, hα4β2, hα4β4 and hα7 expressed in Xenopus oocytes;CHAVEZ-NORIEGA;J. Pharmacol. Exp. Ther.,1997

4. The role of calcium in depolarisation-secretion coupling at the motor nerve terminal;COOKE;J. Physiol.,1973

5. Comparison of neuronal nicotinic receptors in rat sympathetic neurones with subunit pairs expressed in Xenopus oocytes;COVERNTON;J. Physiol.,1994

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