NADH supplementation decreases pinacidil-primed I K(ATP) in ventricular cardiomyocytes by increasing intracellular ATP

Author:

Pelzmann Brigitte,Hallström Seth,Schaffer Peter,Lang Petra,Nadlinger Karl,Birkmayer George D,Vrecko Karoline,Reibnegger Gilbert,Koidl Bernd

Publisher

Wiley

Subject

Pharmacology

Reference27 articles.

1. Enhancement of potassium-sensitive current in heart cells by pinacidil;ARENA;Circ. Res.,1989

2. The co-enzyme nicotinamide adenine dinucleotide (NADH) as biological antidepressant agent. Experience with 205 patients;BIRKMAYER;New Trends Neuropharmacol.,1991

3. Nicodinamide adenine dinucleotide (NADH) - a new therapeutic approach to Parkinson's disease. Comparison of oral and parenteral application;BIRKMAYER;Acta. Neurol. Scand.,1993

4. Therapeutic effects of oral NADH on the symptoms of patients with chronic fatigue syndrome;FORSYTH;Ann. Allergy Asthma Immunol.,1999

5. S-nitroso human serum albumin treatment reduces ischemia/reperfusion injury in skeletal muscle via nitric oxide release;HALLSTRÖM;Circulation,2002

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