Adaptive suppression of the ATF4–CHOP branch of the unfolded protein response by toll-like receptor signalling-Reference-Cited by-同舟云学术

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Adaptive suppression of the ATF4–CHOP branch of the unfolded protein response by toll-like receptor signalling

Published:2009-10-25 Issue:12 Volume:11 Page:1473-1480
ISSN:1465-7392
Container-title:Nature Cell Biology
language:en
Short-container-title:Nat Cell Biol

Author:

Woo Connie W.,Cui Dongying,Arellano Jerry,Dorweiler Bernhard,Harding Heather,Fitzgerald Katherine A.,Ron David,Tabas Ira

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology

Link

http://www.nature.com/articles/ncb1996.pdf

Reference41 articles.

1. Ron, D. & Walter, P. Signal integration in the endoplasmic reticulum unfolded protein response. Nature Rev. Mol. Cell Biol. 8, 519–529 (2007).

2. Zinszner, H. et al. CHOP is implicated in programmed cell death in response to impaired function of the endoplasmic reticulum. Genes Dev. 12, 982–995 (1998).

3. Oyadomari, S. et al. Targeted disruption of the Chop gene delays endoplasmic reticulum stress-mediated diabetes. J. Clin. Invest. 109, 525–532 (2002).

4. Feng, B. et al. The endoplasmic reticulum is the site of cholesterol-induced cytotoxicity in macrophages. Nature Cell Biol. 5, 781–792 (2003).

5. Song, B., Scheuner, D., Ron, D., Pennathur, S. & Kaufman, R. J. Chop deletion reduces oxidative stress, improves β cell function, and promotes cell survival in multiple mouse models of diabetes. J. Clin. Invest. 118, 3378–3389 (2008).

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