Mice with altered KCNQ4 K+ channels implicate sensory outer hair cells in human progressive deafness-Reference-Cited by-同舟云学术

Mice with altered KCNQ4 K+ channels implicate sensory outer hair cells in human progressive deafness

Published:2006-01-26 Issue:3 Volume:25 Page:642-652
ISSN:0261-4189
Container-title:The EMBO Journal
language:
Short-container-title:EMBO J

Author:

Kharkovets Tatjana,Dedek Karin,Maier Hannes,Schweizer Michaela,Khimich Darina,Nouvian Régis,Vardanyan Vitya,Leuwer Rudolf,Moser Tobias,Jentsch Thomas J

Publisher

Wiley

Subject

General Immunology and Microbiology,General Biochemistry, Genetics and Molecular Biology,Molecular Biology,General Neuroscience

Reference46 articles.

1. Longitudinal gradients of KCNQ4 expression in spiral ganglion and cochlear hair cells correlate with progressive hearing loss in DFNA2

2. Differential Expression of KCNQ4 in Inner Hair Cells and Sensory Neurons Is the Basis of Progressive High-Frequency Hearing Loss

3. Deafness and renal tubular acidosis in mice lacking the K-Cl co-transporter Kcc4

4. Loss of K-Cl co-transporter KCC3 causes deafness, neurodegeneration and reduced seizure threshold

5. Regulation of the voltage-gated potassium channel KCNQ4 in the auditory pathway

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