Neurodevelopmental and neuropsychiatric behaviour defects arise from 14-3-3ζ deficiency-Reference-Cited by-同舟云学术

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Neurodevelopmental and neuropsychiatric behaviour defects arise from 14-3-3ζ deficiency

Published:2011-11-29 Issue:4 Volume:17 Page:451-466
ISSN:1359-4184
Container-title:Molecular Psychiatry
language:en
Short-container-title:Mol Psychiatry

Author:

Cheah P-S,Ramshaw H S,Thomas P Q,Toyo-oka K,Xu X,Martin S,Coyle P,Guthridge M A,Stomski F,van den Buuse M,Wynshaw-Boris A,Lopez A F,Schwarz Q P

Publisher

Springer Science and Business Media LLC

Subject

Cellular and Molecular Neuroscience,Psychiatry and Mental health,Molecular Biology

Link

http://www.nature.com/articles/mp2011158.pdf

Reference65 articles.

1. Berg D, Holzmann C, Riess O . 14-3-3 Proteins in the nervous system. Nat Rev Neurosci 2003; 4: 752–762.

2. Fu H, Subramanian RR, Masters SC . 14-3-3 Proteins: structure, function, and regulation. Annu Rev Pharmacol Toxicol 2000; 40: 617–647.

3. Toyo-oka K, Shionoya A, Gambello MJ, Cardoso C, Leventer R, Ward HL et al. 14-3-3Epsilon is important for neuronal migration by binding to NUDEL: a molecular explanation for Miller-Dieker syndrome. Nat Genet 2003; 34: 274–285.

4. Aitken A . 14-3-3 Proteins: a historic overview. Semin Cancer Biol 2006; 16: 162–172.

5. Rosner M, Hengstschlager M . 14-3-3 Proteins are involved in the regulation of mammalian cell proliferation. Amino Acids 2006; 30: 105–109.

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