Reply to ‘TDP43 aggregates: the ‘Schrödinger’s cat’ in amyotrophic lateral sclerosis’
Author:
Publisher
Springer Science and Business Media LLC
Subject
General Neuroscience
Link
http://www.nature.com/articles/s41583-021-00478-0.pdf
Reference8 articles.
1. Tziortzouda, P., Van Den Bosch, L. & Hirth, F. Triad of TDP43 control in neurodegeneration: autoregulation, localization and aggregation. Nat. Rev. Neurosci. 22, 197–208 (2021).
2. Lanznaster, D., Hergesheimer, R., Vourc’h, P., Corcia, P. & Blasco, H. TDP43 aggregates: the “Schrödinger’s cat” in amyotrophic lateral sclerosis. Nat. Rev. Neurosci. https://doi.org/10.1038/s41583-021-00477-1 (2021).
3. Popper, K. Logik der Forschung (Springer-Verlag GmbH, 1935). English translation available in Popper, K. The Logic of Scientific Discovery (Routledge, 1959).
4. Feneberg, E., Turner, M. R., Ansorge, O. & Talbot, K. Amyotrophic lateral sclerosis with a heterozygous D91A SOD1 variant and classical ALS-TDP neuropathology. Neurology 95, 595–596 (2020).
5. Okamoto, Y. et al. An autopsy case of SOD1-related ALS with TDP-43 positive inclusions. Neurology 77, 1993–1995 (2011).
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2. Astrocytes display cell autonomous and diverse early reactive states in familial amyotrophic lateral sclerosis;Brain;2022-01-19
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