TDP43 aggregates: the ‘Schrödinger’s cat’ in amyotrophic lateral sclerosis
Author:
Publisher
Springer Science and Business Media LLC
Subject
General Neuroscience
Link
http://www.nature.com/articles/s41583-021-00477-1.pdf
Reference10 articles.
1. Tziortzouda, P., Van Den Bosch, L. & Hirth, F. Triad of TDP43 control in neurodegeneration: autoregulation, localization and aggregation. Nat. Rev. Neurosci. 22, 197–208 (2021).
2. Feneberg, E., Turner, M. R., Ansorge, O. & Talbot, K. Amyotrophic lateral sclerosis with a heterozygous D91A SOD1 variant and classical ALS-TDP neuropathology. Neurology 95, 595–596 (2020).
3. Jeon, G. S. et al. Pathological modification of TDP-43 in amyotrophic lateral sclerosis with SOD1 mutations. Mol. Neurobiol. 56, 2007–2021 (2019).
4. Sumi, H. et al. Nuclear TAR DNA binding protein 43 expression in spinal cord neurons correlates with the clinical course in amyotrophic lateral sclerosis. J. Neuropathol. Exp. Neurol. 68, 37–47 (2009).
5. Okamoto, Y. et al. An autopsy case of SOD1-related ALS with TDP-43 positive inclusions. Neurology 77, 1993–1995 (2011).
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